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dc.contributor.authorAbasi, M
dc.contributor.authorLotfi, P
dc.contributor.authorBazmani, A
dc.contributor.authorMatini, M
dc.contributor.authorHajilooi, M
dc.date.accessioned2018-08-26T08:54:29Z
dc.date.available2018-08-26T08:54:29Z
dc.date.issued2014
dc.identifier.urihttp://dspace.tbzmed.ac.ir:8080/xmlui/handle/123456789/54146
dc.description.abstractBackground: Several lines of evidence demonstrating that innate and adaptive immunity play important roles in the defense against visceral leishmaniasis (VL). A polymorphism within the Fc?RIIIB gene can lead to the expression of three variants of NA1, NA2, and the combined one (NA1/NA2) which alters affinity of IgG to its receptor.
dc.language.isoEnglish
dc.relation.ispartofIranian Red Crescent Medical Journal
dc.subjectFc gamma receptor IIIb NA1
dc.subjectFc gamma receptor IIIb NA2
dc.subjectimmunoglobulin receptor
dc.subjectunclassified drug
dc.subjectallele
dc.subjectantibody titer
dc.subjectarticle
dc.subjectbinding affinity
dc.subjectcontrolled study
dc.subjectcross-sectional study
dc.subjectDNA sequence
dc.subjectfemale
dc.subjectgenetic polymorphism
dc.subjectgenotype
dc.subjecthuman
dc.subjectimmune response
dc.subjectimmunofluorescence test
dc.subjectmajor clinical study
dc.subjectmale
dc.subjectpolymerase chain reaction
dc.subjectvisceral leishmaniasis
dc.titleEvaluation of Fc?RIIIB-NA1/NA2 Polymorphism in Visceral Leishmaniasis
dc.typeArticle
dc.citation.volume16
dc.citation.issue4
dc.citation.indexScopus
dc.identifier.DOIhttps://doi.org/10.5812/ircmj.12437


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