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dc.contributor.authorZununi Vahed, S
dc.contributor.authorSamadi, N
dc.contributor.authorMostafidi, E
dc.contributor.authorArdalan, MR
dc.contributor.authorOmidi, Y
dc.date.accessioned2018-08-26T05:39:32Z
dc.date.available2018-08-26T05:39:32Z
dc.date.issued2016
dc.identifier.urihttp://dspace.tbzmed.ac.ir:8080/xmlui/handle/123456789/40218
dc.description.abstractChronic allograft dysfunction is the most common cause of allograft lost. Chronic allograft dysfunction happens as a result of complex interactions at the molecular and cellular levels. Genetic and environmental factors both influence the evolution and progression of the chronic allograft dysfunction. Epigenetic modification could be considered as a therapeutically modifiable element to pause the fibrosis process through novel strategies. In this review, the PubMed database was searched for English-language articles on these new areas.
dc.language.isoEnglish
dc.relation.ispartofIranian journal of kidney diseases
dc.subjectAllografts
dc.subjectChronic Disease
dc.subjectEpigenesis, Genetic
dc.subjectFibrosis
dc.subjectGenetic Variation
dc.subjectHumans
dc.subjectKidney Transplantation
dc.subjectKidney Tubules
dc.subjectMicroRNAs
dc.subjectPrimary Graft Dysfunction
dc.titleGenetics and Epigenetics of Chronic Allograft Dysfunction in Kidney Transplants.
dc.typearticle
dc.citation.volume10
dc.citation.issue1
dc.citation.spage1
dc.citation.epage9
dc.citation.indexPubmed


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